• A large Norwegian family with inherited malignant melanoma, multiple atypical nevi, and CDK4 mutation
• A phase I study of the heat shock protein 90 inhibitor alvespimycin (17-DMAG) given intravenously to patients with advanced solid tumors
• A review of inherited predisposition to melanoma
• A systematic screen for CDK4/6 substrates links FOXM1 phosphorylation to senescence suppression in cancer cells
• AXL receptor signalling suppresses p53 in melanoma through stabilization of the MDMX-MDM2 complex
• Biochemical and mutagenic analysis of the melanoma tumor suppressor gene product/p16
• CDKN2A mutations in melanoma families from Uruguay
• Cell cycle control as a promising target in melanoma
• Collagen abundance controls melanoma phenotypes through lineage-specific microenvironment sensing
• Comprehensive insights on genetic alterations and immunotherapy prognosis in Chinese melanoma patients
• Connecting the Dots: Therapy-Induced Senescence and a Tumor-Suppressive Immune Microenvironment
• Detection and quantification of blood-derived CD8+ T lymphocytes secreting tumor necrosis factor alpha in response to HLA-A2.1-binding melanoma and viral peptide antigens
• Detection of novel quantitative trait loci for cutaneous melanoma by genome-wide scan in the MeLiM swine model
• Familial Melanoma and Susceptibility Genes: A Review of the Most Common Clinical and Dermoscopic Phenotypic Aspect, Associated Malignancies and Practical Tips for Management
• Familial risk of melanoma and links with other cancers
• Germline splicing mutations of CDKN2A predispose to melanoma
• Immunohistochemical analysis of the D-type cyclin-dependent kinases Cdk4 and Cdk6, using a series of monoclonal antibodies
• Lack of germline CDK6 mutations in familial melanoma
• Linkage analysis of familial melanoma and chromosome 6 in 14 Australian kindreds
• Melanoma genetics
• MicroRNA-124a is epigenetically regulated and acts as a tumor suppressor by controlling multiple targets in uveal melanoma
• Molecular and genetic mechanisms in melanoma
• Mutation of the CDKN2A 5' UTR creates an aberrant initiation codon and predisposes to melanoma
• p16 proteins from melanoma-prone families are deficient in binding to Cdk4
• p16INK4a-induced senescence is disabled by melanoma-associated mutations
• POT1 loss-of-function variants predispose to familial melanoma
• Signalling responses linked to betulinic acid-induced apoptosis are antagonized by MEK inhibitor U0126 in adherent or 3D spheroid melanoma irrespective of p53 status
• Targeting Translation and the Cell Cycle Inversely Affects CTC Metabolism but Not Metastasis
• The contribution of large genomic deletions at the CDKN2A locus to the burden of familial melanoma
• The genetics of hereditary melanoma and nevi. 1998 update
• The PI3K/AKT signaling pathway controls the quiescence of the low-Rhodamine123-retention cell compartment enriched for melanoma stem cell activity
• Unveiling the genetic landscape of hereditary melanoma: From susceptibility to surveillance